Bronchial asthma attack which repeatedly complications?

(1) bronchitis, "" chronic bronchitis and asthmatic bronchitis attack was the seasonal asthma or allergic asthma not generally often associated with chronic bronchitis, but the resistance of the body such as the lower, or regular contact with irritating gases, Dust or inadvertent catch cold. by the virus may be even the secondary bacterial infection and chronic bronchitis. secretions in patients with bronchial asthma, mucus to remove obstacles, such secretions both airway obstruction. will enable the virus or bacteria have entered contains a great deal of airway secretions arising from secondary infection, In particular, due to a severe attack of asthma often large or long-term use of glucocorticoid drugs there will be even more stubborn and resistant strains of Streptomyces infection. Because elderly patients with secondary bronchitis. easier to form asthmatic bronchitis. General frail or chronic cough will also be transformed into asthmatic bronchitis. (2) emphysema and cor bronchial asthma, bronchial smooth muscle spasm, mucosal edema, increased secretions, so incomplete formation of bronchial obstruction. Getter, inspiratory muscle contraction, thorax expansion, but they can also enter the alveolar gas; breath, closed tracheal collapse, airway resistance was increased further, alveolar gas can not be fully discharged and gradually increase. the whole lung expansion But alveolar elastic fibers and no pathological bronchial smooth muscle damage, this time if the bronchial spasm can be alleviated. Intrapulmonary gas will be discharged, the patient can be fully restored. Therefore, Emphysema is reversible. If long-term repeated attacks, lung tissues can occur following pathological changes : ① bronchi and bronchioles proliferative smooth muscle hypertrophy, bronchiole forming organic stenosis; ② bronchial gland hypertrophy, viscous substances secreted by the bronchioles obstruction; ③ bronchiole cartilage damage, deprived of the normal role of the stent, inspiration, bronchiectasis, has been able to enter the alveolar gas; But breath, bronchial closed subsidence not emit gases, alveolar gas accumulation and swelling; ④ long-term gas concentrations, pulmonary hypertension within the alveolar bloated flexibility diminishes, eventually rupture, leading to the formation of emphysema. If conditions continue to develop, through the following mechanisms may lead to pulmonary hypertension : ① pulmonary artery spasm. Because there hypoxic pulmonary function impairment, hypercapnia, directly lead to pulmonary artery spasm; ② change in pulmonary vascular bed. With the intensification of emphysema, pulmonary hypertension within, oppression alveolar capillary wall, causing luminal stenosis or occlusion, and the alveolar capillary wall rupture caused damage to the network, such as lung capillary bed greatly reduced, increased pulmonary vascular resistance, pulmonary artery pressure increased; ③ sodium and water retention causing increased blood volume also increased pulmonary hypertension. chronic pulmonary hypertension so that the right heart overload occurred hypertrophy, which eventually lead to right-sided heart failure. Treatment is the key to easing bronchial spasm, improving hypoxia and hypercapnia. Only in this way can we effectively reduce pulmonary hypertension and right heart load reduction.