Parasympathetic hyperparathyroidism

Parasympathetic outlined hyperthyroidism is excited by the parasympathetic enhancements arising from a series of symptoms, signs, mainly to slow down the heart rate, blood pressure, syncope, spastic Miosis other. Cause pathogenesis one. Carotid sinus lesions in and around : Local arteriosclerosis, arteritis, neck lymph nodes, tumor, trauma, thyroid surgery, and so on. Normal right carotid sinus stretch sensitive stimulation caused by the feeling of impulse, glossopharyngeal nerve branches across to the medulla, so that the vagus nerve stimulation, reflex caused the heart rate and blood pressure temporary decline. When these lesions, the carotid sinus reflex allergies, In a sudden turn or neck collar tight, and other factors to stimulate the unilateral or bilateral carotid sinus could cause significant pulse slowed, blood pressure decline and even syncope. 2. Pontine hemorrhage, tumor, inflammation, acute purulent meningitis, cavernous sinus infections and orbital lesions, etc. : these lesions to stimulate E-M nuclear, ciliary ganglion or the oculomotor nerve fibers caused oculomotor nerve fibers excited about expanding pupil muscle contraction. Clinical manifestations of carotid sinus syndrome : sudden loss of consciousness, may have vertical dizziness, limb weakness, paleness, cold sweats, nausea, heart rate and low blood pressure. Attack short, the attack can be divided into three types : ① vagal type (heart suppressed) : mainly to bradycardia; ② inhibit vascular type : blood pressure drop; the ③ brain : mainly to loss of consciousness. Pupil spastic reduced light reflection disappeared. Homatropine with eye-pupil can be seen clearly dispersed. A differential diagnosis. Carotid sinus syndrome (cartid sinus syndrome) (1) arteriosclerosis (arte riosclersis) often appear in over 50 years, accompanied by heart, brain, kidney arteriosclerosis performance, such as dizziness, insomnia, memory loss, chest tightness, palpitations, chest pain, such as proteinuria. Fundus examination showed arteriosclerosis. (2) arteritis (artertis) was particularly prevalent in primary aortic arch syndrome. Found in young women, 20 - 30-year-old onset, the distal vascular lesions of low blood pressure, pulse weakened or disappeared, Department of vascular lesions noise carotid artery involvement arise visual impairment, dizziness, syncope, hemiplegia, carotid artery pulse weakened or disappeared, fundus retina anemia. (3) neck mass or neck scar scar or larger mass or see the naked eye, can be mass-hour touch. Cervical lymphadenitis when lymphadenopathy, tenderness, the smooth surface can be driven; Neck metastases quality hardware, border unclear, mobile poor. 2, spastic Miosis (1) cerebellopontine Change : 1. pontine hemorrhage (pons haemorrhage) side of a small amount of bleeding for a comatose performance, hemiplegia; However, the majority involving bilateral, severity of illness, a deep coma, the central high fever, the two-point narrowing the pupil, quadriplegia 1 -2 more died within days. 2. Pontine tumors (tumor pons) onset implicit attack, the slow progress, increased intracranial pressure performance crossover sensation and movement disorders, the general unintentional obstacles. (2) acute purulent meningitis (acute purulent meningitis) rapid onset , high fever, headache, vomiting, meningeal irritation, increased blood leukocytes, neutrophils increased CSF was purulent change. move the eyes of the skull base by stimulated Miosis. (3) the cavernous sinus thrombosis phlebitis (cavernouts sinus thrombophleb itis) Multiple secondary to the face, or orbital infection, often associated with sepsis. Have a fever, headache, conjunctival edema, hyperemia, the earliest Oculomotor, Trochlear nerve. Outreach nerve and the trigeminal nerve stimulation of the first symptoms, the total number of leukocytes and neutrophils increased. (4) Department of orbital lesions orbital thrombosis situation phlebitis, Periostitis oculomotor nerve stimulation appeared Miosis. accompanied by exophthalmos, pain, conjunctival hyperemia and edema, fever, increased leukocyte, neutrophil increased.