Peripheral facial paralysis sequela-what?

⑴ face spastic muscle fibers ipsilateral emerging small and fast, not a constant part of the muscle contraction pull take hold. accompanied blink movement increased. ⑵ facial spasm following features : ◆ facial spasm is a painless, rules-clonic facial muscles twitch, usually first started in orbicularis oculi muscle contraction and spasm often confined to the eyelid or numbness of the mouth, can be expanded to the whole serious hemi-face, including the platysma. ◆ ② often side, the left side than the right side disappearing rarely occur bilaterally. ⑶ facial spasm can be spontaneous, or have free movement in the future, but also because of some facial muscles, such as the talk, eat, chatting precipitated or exacerbated. When the nervous, reading time was too long, fatigue or lack of sleep when symptoms increase, convulsion frequent. And the rest at or emotional stability or reduce the symptoms disappear, sleep twitch stop. ④ each spasm sustained a few seconds to a few minutes, intermittent after successive attacks, the attacks of clear consciousness. ⑤ facial spasm often seen in middle-aged or elderly people, but it rarely occurs in children, women more than men. ⑥ EMG, damaged muscles showed a high frequency of rhythmic motor unit discharge (50 ~ 100 /) ⑶ facial muscles long joint campaign The upper facial nerve paralysis, paralyzed by the side of facial muscles and increase tension eyelid fissile narrow altercation crucifixion, deep nasolabial fold, is a form of pseudo-side muscle facial paralysis, but smiling, still show the true side of facial paralysis. Not fully recovered from the facial paralysis, in addition to the contralateral facial paralysis, POS, hemiplegic be unusual joint campaign. In performance, when the orbicularis oris muscle movement (appetite, said teeth, dark gills, suck), orbicularis oculi muscle also contraction (blepharophimosis smaller). Conversely, when the orbicularis oculi muscle contraction (eyes closed), orbicularis oris muscle also contraction (altercation stretch). The occurrence of this phenomenon is considered to be the mechanism of facial nerve axon regeneration labyrinth. Another argument is that the restoration of the facial nerve myelin bad, personally excited nerve stimulation, neighboring nerve fibers have been exciting phenomenon. ⑷ crocodile tears syndrome patients ate tears, this phenomenon appears in facial nerve palsy after a few weeks or a few months.