Pseudo-mast

Overview muscle hypertrophy, but the elasticity, strength, tendon reflexes weakened claimed pseudo-mast. If muscle hypertrophy and muscle elasticity, strength, tendon reflexes were normal, it was true mast. Cause pathogenesis in pseudo-mast, adipose tissue, fat cells can be seen filling large muscle groups. Terminally ill, very irregular muscle fibers, or even disappear entirely by fat and connective tissue substitutes. False mast was particularly prevalent in the myopathic muscular atrophy, the etiology is unknown, most related to genetic factors. Clinical performance was particularly prevalent in the myopathic muscular atrophy may be true transition from mast. Muscle hypertrophy and muscle elasticity, strength, tendon reflexes weakened, more muscular atrophy at the neighboring site, can cause different sub-distribution is different. In case of muscular dystrophy occurs in the deltoid muscle and the muscle. Differential diagnosis (1) Muscular Dystrophy fake fertilizers large (pseudohy pertrophic muscu deviation dystrophy) is an X-linked recessive genetic, According to the age of onset and the development of disease can be divided into the following two types : 1.Duchenne type : more common in male children. Most walk late, or easy, slow jogging after school, physical education or not, those arising from parents demanding attention. Age of onset for 4 - 8-year-old. Condition gradually worsened, involving pelvic belt, muscle and back muscles, New spinal disease, legs apart, walking in the pelvis next swing. and signs of "duck step." Since sit up again, abdomen, hip, thigh and weak, suffering from said first to switch-prone, and then hands foot support, Equality knee training sequence Pan Fu, can upright, the so-called Cowers levy. As the illness progressed in three-five years to emerge scapular with myasthenia gravis, which dress, leaping movements difficult. Shoulders sag, wings, the former Saw trapezius muscle and not fixed within the margin of scapula, specimens from chicken-wing, "wing-like shoulders." Patients strap muscles, pelvic muscle atrophy. Bilateral gastrocnemius, sometimes arm muscle, shoulder girdle muscular hypertrophy and solid, called false mast. Tendon reflexes disappear, I feel normal. EMG visible action potential duration shortened, reduce volatility, multiphase wave increased, a small amount of production can defibrillation. Some intelligent lower sick child. Serum creatine phosphokinase excited (CPK) increased. 2.Becker type : also known as benign Duchenne muscular dystrophy, often onset after the age of 10, progress has been slow. To begin with the pelvis and thigh muscles atrophy weakness in 5 ~ 10 years involving shoulder girdle and arm. average to 30-year-old has lost walking ability. Patients were advised at the gastrocnemius muscle hypertrophy. Pathology, EMG, CPK and other broadly with Duchenne. (2) of congenital muscular rigidity (myotonica congenita) found in early childhood. for autosomal dominant inheritance. The main clinical features of systemic muscle rigidity and mast. Performance of activities for the physically rigid. Benzene exhumation movement, static rest or cold symptoms add to the environment. Children often eat when I first opened his mouth after chewing and not, stayed behind to stand up immediately after, unable to stand after the start. Handshake not immediately relax, laugh after the expression can not immediately recover muscle caused plenty of amazement. Not even fall when a hand to support the above symptoms were repeated after exercise to reduce or disappear. Checks can be seen patients with systemic skeletal muscle hypertrophy, tapping local muscle can produce depression or muscle the ball, slowly dissipated. General feeling normal. EMG can see a contraction caused a series of action potentials.