Hypoglycemic coma

Hypoglycemia outlined for biochemical abnormalities, is not a disease. All for one reason or another so that the normal blood sugar dropped to below sympathetic to the cause excitement and central nervous system dysfunction due to the performance of a clinical performance known as hypoglycemia. The disease can lead to serious coma. Cause pathogenesis of hypoglycemia caused many reasons, can be broadly classified under the following categories. (1) too much insulin : Insulin tumor, islet cell proliferation, islet cell carcinoma, ectopic insulin-secreting tumor, antidiabetic drugs. Clinical endogenous or external insulin-induced hypoglycemia most common. (2) reactive hypoglycemia : Early diabetes, functional hypoglycemia, nutrition hypoglycemia. (3) excessive sensitivity to insulin. (4) liver disease. (5) poisoning : drug poisoning. (6) lack of carbohydrates. Clinical manifestations of hypoglycemia on the body is a strong stress, sympathetic performance patients excited; Hypoglycemia central nervous system so that the lack of energy sources, there are many dysfunction. McGREGOR early onset patients dizziness, headache, hunger, weak and physically damp and cool, hazy sense of the forest, orientation obstacles, convulsion and even coma, may also be involved to be suffering from mental disorder and hemiplegia. Diagnosis of blood glucose results. Treatment (1) general treatment : to determine whether patients with airway patency and, if necessary, do the same; Fits tongue when necessary to prevent injury. (2) emergency treatment : patients are still swallowing movements, they can feed some dessert, the majority can quickly improve the symptoms. Coma should have immediate intravenous glucose, a per-minute rate of 10 ml 50% glucose injection of 50 ml. For the majority of patients with 20% glucose-60ml50 to correct hypoglycemia. In the rapid injection of glucose, can produce symptomatic hypokalemia. Most of hypoglycemia patients after injection of glucose within 5 ~ 10min can awakenings. If severe hypoglycemia, lasted longer, nerve function is not a very long time to recover. Awake after the early juice and food consumption. In patients use insulin (low precision fish protamine zinc insulin), or C urea chlorosulfophenolazo may have hypoglycemia reaction, of these patients, after sober again to prevent hypoglycemia reaction, the need to observe 12 ~ 48 hours. Sometimes eating carbohydrates at the same time, plus high-protein diet such as milk and static point 5% glucose is necessary.