Patellofemoral clonic ankle clonus

Cause pathogenesis patellar clonic and ankle clonus tendon reflexes is highly enhanced features, They can occur at any increased tendon reflexes occasions, including the nervous system without organic lesion. In neurosis and systemic physiological hyperreflexia of jerks and organic diseases of jerks, the former is usually not constant, both sides of the same general level of performance that is not associated with organic symptoms. 2 reflection asymmetry, said they were organic diseases. In the spinal cord or nerve root in the gray matter of reflex arc damage, or side reflex, indicates pyramidal tract damage. Clinical manifestations patient supine, leg extension, inspection of thumb used by two food means clamping curbs on the patella, to the bottom of a sudden promotion and maintain a sense, attached to the patella on the edge of the quadriceps tendon elongated, when the knee jerk caused the increased muscle contraction continue to extend tendon, patella, that is, continuous, under the rhythm of its viewers. The ankle clonus inspection Waterloo entrusted with the left and right hand front foot at first light to the Ministry of upward buckling, and we have not proved to make lateral plantar flexion, The foot was then alternating the next 510282 fibrillation. Differential diagnosis (1) toxic encephalopathy (toxic encephalopathy) poisoning history, more slow onset, orientation obstacles, forgotten, euphoric, mental retardation, ophthalmoplegia, or associated with ataxia, tremor, Some with retinal hemorrhage, nystagmus, hallucinations and paranoia, a sense of ambiguity or even coma. accompanied by pyramidal tract damage appeared patellar clonic and ankle clonus, limbs increased muscle tone, pathological positive. (2) virus encephalitis (viral encephalitis) acute onset, generally have fever, disease before flu on history and are vomiting, unconsciousness and spirit. There will be a state of delirium, drowsiness and coma, hemiplegia, Mr paralysis, aphasia, hemiplegia sensory dysfunction, and seizures pyramidal tract symptoms, there patellar clonic and ankle clonus, accompanied by myoclonus, tremor, Athetosis disease. Brainstem involvement when there cranial nerve paralysis, bilateral limb paralysis and sensory dysfunction, ataxia, respiratory and circulation disorders. Meningeal irritation, increased pressure on the lumbar puncture, cerebrospinal fluid increased leukocyte. (3) familial spastic paralysis (familial spastic paralegia) more than before onset in children, was conducted, the performance of the two lower extremities, motor neuron injury signs, and some are gradually reached upper extremity. Paralysis of the limbs increased muscle tone, spastic gait, tendon hyperreflexia, appeared on the side of the patellofemoral clonic and ankle clonus. Pathological positive reflection. Most patients had segmental foot. No sensory impairment, mild ataxia. Some patients may have a language barrier. Cerebral cortex involvement can be a smart low. (4) acute myelitis (acute mgelysis) rapid onset, more than a few hours to a week to peak. 1 ~ 2 weeks before infection. If the spinal cord damage occurs tetraplegic, tendon reflexes disappear, pathological reflex not leads. 3 ~ 4 weeks later, with spinal shock disappeared, and the limbs flaccid paralysis from the transition to a spastic paralysis, increased muscle tone, Tendon hyperreflexia, there ankle clonus and patellar clonic, pathological reflex for the positive. Involving the respiratory muscles appeared difficulty breathing, coughing inability. Often back pain or abdominal pain and chest constrictions flu, all of the following lesions deep feeling deletion in the early urine retention.