Monday, March 10, 2008

Hoffmann's sign

Cause pathogenesis of the ulnar nerve afferent nerve, median nerve, spinal cord centers in 6 -8, a thoracic and efferents to the median nerve, Cone beam damage this reflex unilateral or bilateral intensity asymmetry or accompanied by other signs of pyramidal tract injury. Spinal cord lesions, cerebral arteriosclerosis, peripheral nerve damage, Neurosis, neural excitability increased existence of this reflex, most of symmetry, the pyramidal tract without other signs. Clinical manifestations examiner to right index and middle finger gripper with the middle finger tip their wrist dorsiflexion. refer to the rest of the buckling relax in a natural state. Examiners to quickly thumb missiles pointed fingernails scratching patients, at this time, given the claim by the sudden deep flexor stretch, caused the buckling refers hand reflection is positive, if the positive side, the side tendon hyperreflexia, Cone beam damage suggest, as both positive and there is no neurological signs no positioning sense. Differential diagnosis (1) neural-miya Disease (neurosis) have more spirit, more females than males. Early many insomnia, headache, anxiety, distracted, general malaise, accompanied by nausea, palpitation, neurasthenia and other symptoms, In serious cases, there is more dream, memory decline, depression, and believes that it is a serious incurable disease. No nervous system, the positive signs, various kinds of examinations are normal, there will be a bilateral Hoffman levy positive. (2) amyotrophic lateral sclerosis (amyotrophic lateral sclerosis) In more than 40 years old onset, as lesions involving the anterior horn cells and pyramidal tract, Bilateral and emerging side of the hand or clumsy and weak, big and small fish thenar muscle, bone interosseous muscle, muscle atrophy Lumbrical obvious, and a "claw hand table." Myasthenia Gravis and muscular atrophy gradually to the forearm, upper arm and shoulder extension, the location of involvement band fibrillation. Because pyramidal tract damage arising increased muscle tone, tendon hyperreflexia, Hoffman levy and the levy was pathological. Lower motor neuron injury pyramidal tract symptoms were covered limb muscle hypotonia, or reduce tendon reflexes disappear. Mostly lower extremity spastic paralysis, increased muscle tone, tendon hyperreflexia, pathological patients. 1 ~ 2 years later, there will be the articulation unclear, the pungent drinking water, dysphagia. (3) spinal cord injury (injury of the spinal cord) injury to the physical plane following flaccid paralysis, muscular tension disappear, feeling and reflection disappeared, sphincter dysfunction, incontinent, called spinal shock. 2 ~ 4 weeks after the disappearance of shock, paralyzed limbs hypermyotonia, knee tendon hyperreflexia, Hoffman levy and bilateral Babinski sign, Cadau grams levy positives. (4) internal carotid artery occlusion (occlusion of iaternal carotidarte y) typical clinical manifestations of the contralateral hemiplegia, face and arm at heart, sensory dysfunction lighter, associated with ipsilateral visual impairment or loss. The latter with the involvement of the ophthalmic artery. Internal capsule after 2 / 3, thalamus, visual, hearing radiation, the globus pallidus medial nuclear structure of choroidal artery involvement. there will be the opposite with the Blind to feel excessive, thalamus hands. Contralateral lesions increased muscle tone, tendon hyperreflexia, Hoffman levy positive Babinski sign and Card Road grams levy positive.

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