Saturday, March 1, 2008

High blood lipids and what is the relationship between brain

Although the occurrence of high blood lipid cerebrovascular disease risk still not sure, but led to high blood lipids artery intimal lipid calm, may accelerate the formation of atherosclerosis that fact, it could be considered too high lipids and the incidence of stroke is related. Lipid high blood means one or more abnormal lipid composition of the increase. Prosperous people know intake of high-fat and high-cholesterol diet-induced atherosclerosis, in part of a genetic defect lipid metabolism of performance obviously, these people because of the difficulty in removing blood. Cycle neutral excessive fat and cholesterol, or have too much body, which leads to blood and guts solid opponent triglyceride concentrations increased. In recent years the diet of fat and atherosclerosis has made a new understanding. Alaska's Eskimo North America, although a lot of fat intake diet, but they arteriosclerosis of the lower incidence. They like eating a lot of marine organisms, such as seashells, oysters and caviar, the study found that the marine biological fat content of more than 20% is carbon 20 5 acid, which the body of the double-stranded with three prostaglandin I2 (PGI3) and thromboxane A2 (TXA3). General fat and lard as linoleic acid (18 carbon acid 2), and peanuts high acid content, which in the body into two double bond with the prostate (PGI2) and thromboxane (TXA2). TXA3 and TXA2 different, it does not cause the release of platelet aggregation active substances, carbon 20 5 acid may protect blood vessels and thrombosis occurred is not easy. The vegetable oil fatty acid linolenic acid is mainly (18 carbon 3 acid), in vivo may be extended to carbon chain saturated and the generation of carbon 20 5 acid, a pair of vascular protection. Right blood lipid including various lipid cholesterol, triglyceride and phospholipid composition of the analysis that if the lipid polyunsaturated fatty acids were higher volume is difficult occurrence of atherosclerosis. Such evidence can we inspire one-sided emphasis on limiting high fat intake, because a number of essential fatty acid intake on the body is beneficial. According to Japanese scholars in the spontaneously hypertensive rats developed a risk of stroke in spontaneously hypertensive rats, rats in these statements to contain 20% fat, and various quantitative cholesterol diet for one group, another group including 4.5% fat, 17% protein, 58.6% of carbon water compounds and salt O.39% of the standard diet. Results of the former, that is, to high fat, high-cholesterol diet of a group, serious hypertension and stroke incidence of anti-fell. With some scholars have found lower serum cholesterol levels in areas with higher incidence of stroke views are consistent. The exact mechanism yet, but at least we can see that cholesterol is not the people that bad. Intestinal absorption of cholesterol was, after liver complex metabolic process and enter the blood, cholesterol and protein when combined together to form lipoproteins. Lipoprotein three, according to particle size of the order as very low-density lipoprotein, low-density lipoprotein and high-density lipoprotein. Infiltration of the vascular wall and cause atherosclerosis is very low density lipoprotein and low density lipoprotein and high-density lipoprotein shoulders to calm the vascular wall cholesterol "separated" from the liver and go back to the "cleaning" tasks, high density lipoprotein is very important. It is reported that in patients with cerebrovascular disease, high density lipoprotein, the reduction of animals (susceptibility to stroke and hypertension in rats) fed high-fat, high-cholesterol food, the blood pressure drop, necrosis of the small artery lesions was inhibited, cerebrovascular disease incidence declines in the results is consistent. Modest containing more unsaturated fatty acids in the diet is reasonable.

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