Leukoplakia

Outlined by the oral mucosal epithelial metabolic disorder, there hyperkeratosis, keratosis white plaque there, it said leukoplakia. More common in middle-aged men. Case white minority carcinogenesis may occur. Etiopathologic etiology and pathogenesis is not yet very clear, but the clinic often seen with the following factors : smoking, After the burning of tobacco nicotine and the mucosa of thermal stimulation. Others such as chewing betel nuts, wine, vinegar, hot, horrified, from bad repair, dental esthetics, residual roots and crown of the sharp edge of the mucous membrane such as mechanical stimulation could induce the disease. In addition, animal experiments show that a lack of vitamin A can cause epithelial hyperkeratosis. Candida albicans infection, iron deficiency anemia, vitamin B12 and folate deficiency, also on. Clinical manifestations generally no symptoms, such as epithelial keratosis is heavy, rough, dry mouth, or eating boring, and other symptoms. Good location for the cheek, tongue, lips, palate and other places mucosa (Map 9, 10). More checks were found. Lesions were milky white plaque, a clear boundary slightly higher for mucosal surface, early fashion smooth, gradually expanded, thickening, rough, and the emergence fragmented, deprived of the normal mucosa of flexibility and softness. If a certain part of lesions significantly gray and was verrucous or that there is erosion, ulcers, or base there sclerosis, for biopsy. to exclude the possibility of malignant transformation (Map 11). Map 9 leukoplakia (cheek) Map 10 leukoplakia (dorsum of the tongue)