What are the acute glomerulonephritis complications?

Acute phase of serious complications mainly the cycle of serious congestive state, hypertensive encephalopathy and acute renal failure. As in recent years to strengthen its work on the prevention of incidence and mortality has decreased significantly. 1. Cycle congestive state: because Shuinazhuliu, blood volume dried up, until pulmonary edema. Each reported incidence of varying severity and treatment related. China 50 to 60 years reported to be hospitalized children with acute nephritis 24% to 27% of such complications seen, in recent years, the report has been lowered to 2.4%. Occurred after the onset of acute nephritis 1 ~ 2 weeks. Clinical manifestations of dyspnea, not supine, chest tightness, cough, lung wet at the end of rales, the liver tenderness, such as law Benma about heart failure symptoms, the result of blood volume expansion due to cardiac pump with real exhaust different. At this time of a stroke more often and are not reduced, the normal cycle time, moving no more blood oxygen difference, and the tonic effect of poor digitalis, diuretics and often to ease the application. Very few can be developed to truly severe heart failure, in a few hours to 1 to 2 days rapidly and life-threatening pulmonary edema. 2. Hypertensive encephalopathy: that blood pressure (DBP in particular) dramatically increased, in terms of a central nervous symptoms. General children than adults. Generally believe that this disease is systemic hypertension on the basis of resistance to the brain caused cerebral vasospasm small hypoxic brain edema caused, but also some people believe that the rapid rise of blood pressure, cerebral vascular original with the automatic control systolic and diastolic function of regulation, a high degree of cerebral vascular congestion, a result of cerebral edema, acute nephritis In addition the Shuinazhuliu also play a role in pathogenesis. Occurred early in the course of acute glomerulonephritis, a general acute onset showed severe headache, frequent nausea and vomiting, followed by visual disturbances, vertigo, diplopia, temporary and Mongolia, and lethargy or irritability, if not promptly Treatment occurred convulsions, coma, a few temporary hemiplegia aphasia, when severe cerebral hernia. Nervous system have no limitations signs, and shallow reflection tendon reflexes can weaken or disappear, sometimes positive ankle clonus, and there will be pathological reflex, can be a serious signs and symptoms of cerebral hernia. Fundus examination common retinal artery spasm, and sometimes that papilledema. CSF Qingliang, pressure and protein in normal or slightly increased. Such as blood pressure more than 18.7/12.0 kPa (140/90mmHg), with a visual impairment, seizures and coma of a three could be diagnosed. 3. Acute renal failure: a significant portion of patients with acute nephritis in the acute phase with varying degrees of azotemia, but progress to acute renal failure, only a very small number. Lack of complications is still effective preventive measures, acute nephritis has become the leading cause of death. Clinical manifestations oliguria or urine, blood urea nitrogen, serum creatinine increased, hyperkalemia, metabolic acidosis. Oliguria or urine for three to five or more than one week, after urine increased, the symptoms disappeared, and the gradual return of renal function. 4, secondary bacterial infection, acute nephritis as lower body resistance and easy to secondary infection, the most common being the lungs and urinary tract infection, in case of a secondary infection, it should actively symptomatic treatment, so as to avoid the original disease heavier.