Cirrhosis how this will affect coagulation function?

Normal nasal, oral, gastrointestinal mucosa is not installed their own bleeding, the main body is a good coagulation factor at play. These coagulation factors are generated by the liver. These coagulation factor short life, death fast, timely synthesis of the liver needs new, continue to supply added. When cirrhotic liver cell congestion, edema, fibrosis, sometimes accompanied by liver cells in ischemic necrosis of the liver cell necrosis, the more possible area, the greater this function on the poorer, so that the level of coagulation factor dropping rapidly. Portal hypertension congestive spleen, swelling, hyperthyroidism, platelet integrity has been seriously undermined, not only greatly reduce the number of hemostatic function are significantly lower, so the loss of platelets due to the coagulation function. Systemic capillary fragility, poor liver function, blood coagulation factor of synthetic materials dyspepsia, lack of absorption, which in turn inhibited the growth of the coagulation factor and synthesis. These cirrhosis of these adverse factors, a common role in the body, forming a chronic hemorrhage more than a phenomenon.