How to treat hepatorenal syndrome?

The symptoms certainly effective date no special treatment. Since the serious liver disease can not be alleviated, Its pathogenic factor is always there, nephropathy natural unlikely to ease. Once developed into a typical liver with renal syndrome (also qualitative or hyperlipidemia), but few survivors. Therefore:
(1) for primary liver disease, and actively improve liver function become very important. Most of this syndrome patients died of liver failure staff to stay and portal hypertension, only improve liver function, renal function was expected to be restored.
(2) to have excessive diuretic, large or multiple release ascites and bleeding, dehydration, and other factors caused the decrease in blood volume, or hemodynamic Pai high resistance was low-patients can be tested for expansion, such as the sugar used dextral research, albumin, plasma, and whole blood transfusion and so on its own ascites. But the daily volume of liquid should strictly control the importation in 1000 ml within.
(3) diuresis and improve renal blood flow. Early application of drugs to improve renal blood flow, especially in the early fight for quality hyperlipidemia, or even earlier application may have a certain effect. If the application of alkali - pao Valley (6542) daily 30 ~ 60 ml 10% glucose solution intravenous infusion or intravenous injection at the treatment of severe hepatitis (manic-patient application in the East by 03 ~ 0.9 mg intravenous injection, in the ~ 3 times daily); or dopa face 180 mg / 1,000 Galouzeau furosemide 10 mg / kg intravenous drip, have a certain effect. Multi-bus or peritoneal cavity injection directly to lower portal pressure diuresis. Others adopt unilateral lumbar sympathectomy closed and prostaglandin income, prostaglandin E1 treatment to be effective.
(4) diuresis. Thousands of items available diuretics sigh of plastic each 30 mg or furosemide. Some people think that in the early acute renal failure using high-dose furosemide, can prevent the development of acute renal failure may also oliguria converted to non-oliguric state: It was also reported that the oxygen Jun amine in cirrhotic ascites reduced renal perfusion patients can produce 12 hours of diuretic effect of increasing.
(5) dialysis therapy. Both hemodialysis or peritoneal dialysis, can increase the survival rate of patients with uremia, so this law applies only to liver disease patients is expected to resume.
(6) implantation Lereeu of a trip peritoneal shunt, or liver transplantation, some people estimate that this may be the most promising therapy.
(7) All of the incentives lead to renal failure, such as gastrointestinal bleeding, excessive diuretic, and repeatedly put massive ascites, serious infections. Prohibited renal toxicity of drugs including antibiotics-amino sugar, Neomycin and prostaglandin synthesis inhibitors such as indomethacin, and other non-steroid anti-inflammatory analgesic drugs. Preventing electrolyte disorders, such as hypokalemia and hyperkalemia.
(8) general supportive therapy should be dealt with acute renal failure, according to physiological needs replenishment of people, we must guard against excessive or inadequate, and paid attention to the maintenance of electrolyte and acid-base balance.
In addition, some people reported using peptide vasopressin, between hydroxylamine, captopril acid, a small amount of ascites repeatedly caving, adrenal cortical hormones and doors, such as a shunt to effective treatment.